Diet

Do Overweight People Eat LESS Often?

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Apparently yes according to a new study.

Overweight adults eat less often than people in the normal body weight range, but still take in more calories and are less active over the course of the day, according to a U.S. study. 

By contrast, normal weight adults, including those who had lost a lot of weight and kept it off, ate more often, according to findings published in the Journal of the American Dietetic Association.

“Most of the research has shown that people who eat more frequently have a lower weight. But no one knows why,” said lead researcher Jessica Bachman, an assistant professor in the department of Nutrition and Dietetics at Marywood University in Scranton, Pennsylvania.

More than 60 percent of U.S. residents are obese or overweight, but the relationship between the number of meals people eat each day and the ability to maintain weight loss has remained unclear, she said.

I do know that when I weighed over 300 pounds I did not eat breakfast but would binge eat one or two times during the day. Whereas, today, when I weigh about 240 and want to lose an additional 50 pounds, I eat more often and measure the calories carefully.

The level of satiation is no different, although I do know now when I need some protein.

On average, the normal weight subjects ate three meals and a little over two snacks each day, whereas the overweight group averaged three meals and just over one snack a day.

Generally, though, weight loss “maintainers” consumed the fewest calories, at about 1,800 a day, compared with the normal weight and overweight subjects, who took in 1,900 and more than 2,000 calories a day, respectively.

Bachman said that snacking might help prevent weight gain by staving off intense hunger.

 

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Diet

Want to Lose Weight? Don’t Tell Anyone!

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Anita Mills and her 200 pound weight loss

I am happy for Anita, but this method doesn’t work for me.

Anita Mills was 382 pounds when a family doctor gave her four simple rules to lose weight:

1. Eat 8 ounces of food every 3 hours

2. No sugary drinks

3. Do not skip meals

4. Do not tell anyone what you’re doing

Now 242 pounds lighter, Mills credits that last tip for helping her through the most difficult months of her weight loss journey. Not having someone questioning every bite or trying to persuade her to relax on weekends helped her focus on the goal.

“It’s so much better to walk into a room and have someone say, ‘Hey, did you do something different?’ than to announce, ‘I’m on a diet,’ and have people pointing fingers at you,” she said.

The advice seems counterintuitive. Weight Watchers and similar groups tout support as a major reason for their programs’ success, and studies have found that accountability is important in accomplishing a goal. But telling family, friends and Facebook about your diet plans could have a detrimental effect, some experts say.

I, too, have lost a large amount of weight and have taken many years to do so. I now weigh 240 pounds down from 370 plus or so (8 years ago).

I understand about friends and acquaintenances making comments, but I have found accountability to my wife, children and friends to be a good motivator. Now, I am using MyFitnessPal.com and share my daily diet and exercise with certain friends, who also wish to lose weight.

Different weight loss methods for different people……

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Diet

Vitamin E Supplements May Increase Risk for Prostate Cancer?

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YES, according to a new study.

Men who take a daily vitamin E supplement — a regimen once thought to reduce cancer risk — face an increased risk of prostate cancer, according to results of a large national study.

The finding comes from a report summarizing the latest results of the Selenium and Vitamin E Cancer Prevention Trial (SELECT). Eric Klein, M.D., chair of the Glickman Urological and Kidney Institute at Cleveland Clinic, is the lead author.

SELECT began in 2001 to test earlier research suggesting selenium and vitamin E supplements may reduce the risk of developing certain cancers. Some vitamin supplements containing enhanced levels of selenium and vitamin E were marketed to consumers during this time period with claims of reducing cancer risk.

The paper, which will appear in the October 12 edition of the Journal of the American Medical Association, found that a group of men taking a daily dose of 400 IU of vitamin E from 2001 to 2008 had 17 percent more cases of prostate cancer than men who took a placebo.

“For the typical man, there appears to be no benefit in taking vitamin E, and in fact, there may be some harm,” said Dr. Klein, an internationally renowned prostate cancer expert who served as the national study coordinator.

Prostate cancer is the most common non-skin cancer in American men with a current lifetime risk of 16 percent. For 2011 it is estimated 240,000 new cases and 33,000 deaths will result in the U.S.

Well, I am not taking a vitamin E supplement and will not start now. I am taking a vitamin D supplement, but will discuss continuing this with my physician at my annual physical  exam next month.

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Diet

Want to Ditch the Twinkies and Other High Caloric Food – Feed Your Brain?

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Differences between euglycemic and hypoglycemic conditions.

Axial slices with (A) whole group, covaried for BMI (n = 14), (B) obese group (n = 5), and (C) nonobese group (n = 9) averages, showing brain response to euglycemia compared with mild hypoglycemia across visual cue tasks (threshold of P < 0.05, 2 tailed, FWE whole brain corrected). Red and yellow areas show greater activity during euglycemia, and blue areas indicate greater activity during hypoglycemia. The color scale gives the t value of the functional activity. Eu, euglycemia; Hypo, hypoglycemia; NAcc, nucleus accumbens; Hyp, hypothalamus; VMPFC, ventromedial prefrontal cortex; Hipp, hippocampus; L, left; R, right. MNI coordinates were used to define brain regions.

Yes, according to a new study.

If the brain goes hungry, Twinkies look a lot better, a study led by researchers at Yale University and the University of Southern California has found.

Brain imaging scans show that when glucose levels drop, an area of the brain known to regulate emotions and impulses loses the ability to dampen desire for high-calorie food, according to the study published online September 19 in The Journal of Clinical Investigation.

“Our prefrontal cortex is a sucker for glucose,” said Rajita Sinha, the Foundations Fund Professor of Psychiatry, and professor in the Department of Neurobiology and the Yale Child Study Center, one of the senior authors of the research.

The Yale team manipulated glucose levels intravenously and monitored changes in blood sugar levels while subjects were shown pictures of high-calorie food, low-calorie food and non-food as they underwent fMRI scans.

When glucose levels drop, an area of the brain called the hypothalamus senses the change. Other regions called the insula and striatum associated with reward are activated, inducing a desire to eat, the study found. The most pronounced reaction to reduced glucose levels was seen in the prefrontal cortex. When glucose is lowered, the prefrontal cortex seemed to lose its ability to put the brakes upon increasingly urgent signals to eat generated in the striatum. This weakened response was particularly striking in the obese when shown high-calorie foods.

“This response was quite specific and more dramatic in the presence of high-calorie foods,” Sinha said.

“Our results suggest that obese individuals may have a limited ability to inhibit the impulsive drive to eat, especially when glucose levels drop below normal,” commented Kathleen Page, assistant professor of medicine at the University of Southern California and one of the lead authors of the paper.

A similarly robust response to high-calorie food was also seen in the striatum, which became hyperactive when glucose was reduced. However, the levels of the stress hormone cortisol seemed to play a more significant role than glucose in activating the brain’s reward centers, note the researchers. Sinha suggests that the stress associated with glucose drops may play a key role in activating the striatum.

“The key seems to be eating healthy foods that maintain glucose levels,” Sinha said. “The brain needs its food.”

The abstract for the paper:

Obesity is a worldwide epidemic resulting in part from the ubiquity of high-calorie foods and food images. Whether obese and nonobese individuals regulate their desire to consume high-calorie foods differently is not clear. We set out to investigate the hypothesis that circulating levels of glucose, the primary fuel source for the brain, influence brain regions that regulate the motivation to consume high-calorie foods. Using functional MRI (fMRI) combined with a stepped hyperinsulinemic euglycemic-hypoglycemic clamp and behavioral measures of interest in food, we have shown here that mild hypoglycemia preferentially activates limbic-striatal brain regions in response to food cues to produce a greater desire for high-calorie foods. In contrast, euglycemia preferentially activated the medial prefrontal cortex and resulted in less interest in food stimuli. Indeed, higher circulating glucose levels predicted greater medial prefrontal cortex activation, and this response was absent in obese subjects. These findings demonstrate that circulating glucose modulates neural stimulatory and inhibitory control over food motivation and suggest that this glucose-linked restraining influence is lost in obesity. Strategies that temper postprandial reductions in glucose levels might reduce the risk of overeating, particularly in environments inundated with visual cues of high-calorie foods.

It is common knowledge in running endurance training (e.g. marathon training) that you need to maintain blood glucose levels in order to avoid “negative thoughts” from the brain. Thoughts that will persuade you to stop running.

So, why should this be any different throughout the day or evening when we are not stressing our bodies?

Interesting how obesity plays a role, where an obese person sort of loses their way with regulating their desires for high calorie food.

The entire paper is here. The PDF file is here

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Alzheimer's Disease

Does Diabetes Increase Alzheimer’s Disease Risk?

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Yes, and other forms of dementia as well.

People with diabetes are at increased risk of having a heart attack or stroke at an early age, but that’s not the only worry. Diabetes appears to dramatically increase a person’s risk of developing Alzheimer’s disease or other types of dementia later in life, according to a new study conducted in Japan.

In the study, which included more than 1,000 men and women over age 60, researchers found that people with diabetes were twice as likely as the other study participants to develop Alzheimer’s disease within 15 years. They were also 1.75 times more likely to develop dementia of any kind.

“It’s really important for the [public’s] health to understand that diabetes is a significant risk factor for all of these types of dementia,” says Rachel Whitmer, Ph.D., an epidemiologist in the research division of Kaiser Permanente Northern California, a nonprofit health-care organization based in Oakland, California.

Whitmer, who studies risk factors for Alzheimer’s but wasn’t involved in the new research, stresses that many questions remain about the link between diabetes and dementia. The new study was “well done” and provides “really good evidence that people with diabetes are at greater risk,” she says, “but we really need to look at other studies to find out why.”

More studies are needed and it makes sense that the longer you live, the more of a chance you will develop some sort of dementia. But, it also makes sense that via diet and exercise, you should avoid diabetes in the first place.

Diabetes could contribute to dementia in several ways, which researchers are still sorting out. Insulin resistance, which causes high blood sugar and in some cases leads to type 2 diabetes, may interfere with the body’s ability to break down a protein (amyloid) that forms brain plaques that have been linked to Alzheimer’s. High blood sugar (glucose) also produces certain oxygen-containing molecules that can damage cells, in a process known as oxidative stress.

In addition, high blood sugar — along with high cholesterol — plays a role in the hardening and narrowing of arteries in the brain. This condition, known as atherosclerosis, can bring about vascular dementia, which occurs when artery blockages (including strokes) kill brain tissue.

“Having high glucose is a stressor to the nervous system and to the blood vessels,” says David Geldmacher, M.D., a professor of neurology at the University of Alabama at Birmingham. “The emerging information on Alzheimer’s disease and glucose shows us that we do need to remain vigilant on blood sugar levels as we get older.”

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